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Alopecia areata is noncontagious or not contagious. It occurs with greater regularity in individuals who have affected loved ones suggesting that heredity might be a cause. Strong evidence that genes may increase peril for alopecia areata is discovered by studying families with several affected members. This study identified no less than 4 regions within the genome that are likely to contain alopecia areata genes. In addition it is somewhat more prone to occur in individuals who have relatives with auto immune diseases.

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The condition is believed to be an autoimmune disorder where the natural object attacks its hair follicles and suppresses or stops growth of hair. There is evidence that T cell lymphocytes cluster around these follicles causing excitement and subsequent thinning hair. An unknown environmental trigger like emotional stress or possibly a pathogen is thought to combine with inherited genes to cause the condition. At that place there are some recorded cases of babies being born with congenital alopecia areata, however these are not cases of autoimmune disease because an infant is born without a fully developed disease fighting capability.
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